Šećer iz voća u razumnim količinama sigurno nije problem ali njime zadovoljavati veći dio dnevnih potreba za kalorijama mislim da je veliki problem.
Sam dr Graham je dokaz da je to tako. No ujutro mi se voće čini kao najbolji način za započeti dan. Potiče sokove, detoksificira, a ne opterećuje organizam... za sad se toga držim.
Pridružen/a: 11. 09. 2007. Postovi: 12892 Lokacija: Zagreb
Postano: 18.8.2015. uto. 01:02 Naslov:
ogi je napisao/la:
Kažu da je jetri do 25g optimum, 50g maksimum, 100g ekstrem fruktoze dnevno.
Isto vrijedi za fruktozu i alkohol, možda se čak i zbraja!
Citat:
A study which determined the risk level for developing cirrhosis in Australian men who drank alcohol found the risk increased significantly when alcohol intake exceeded 40 grams per day. The risk for women was determined to occur at a similar intake level. 40 gms/day (4 standard drinks) was concluded to be the safe maximum level for both men and women. [Batey R et al Med J Aust (1992) 156 (6)].
1% of deaths for 1986 were examined in the U.S. Quantity and frequency of alcohol consumption was obtained from each descendant's next of kin. The percentage of deceased with cirrhosis increased sharply with increasing number of drinks per day. An intake of three alcoholic drinks per day was associated with a significantly higher percentage of cirrhosis deaths compared with lifetime abstainers. [Parrish K et al, J Stud Alcohol (1993) 54(4)].
156 papers were reviewed assessing the relation of individual alcohol consumption to risk of physical damage. Evidence was found for a dose-response relationship between alcohol consumption and risk of liver damage. At levels of more than 20-30 grams alcohol/day, all individuals are likely to accumulate risk of harm. [P.Anderson et al. Addiction (1993) 88(11)].
In a consecutive autopsy series of 210 Finnish males, the effects of long-term moderate alcohol consumption on the incidence of liver disease were observed. Below 40 gms of alcohol/day no significant increase in the features of liver disease were apparent. Daily intake of between 40 - 80 gms/day increased the frequency of fatty liver and slight alcoholic hepatitis. The incidence of liver cirrhosis increased significantly when daily intake exceeded 80gms. [V.Savolainen et al. Alcohol Clin Exp Res (1993) 17 (5)].
A Danish study measured the prevalence of abnormal liver-derived enzymes in a population sample of 905 men and women aged 30-50. 12% of the cohort was found to have raised levels of abnormal liver-derived enzymes associated with moderate (48gms/day) alcohol intake. With higher alcohol intake (>48gms/day) the odds ratio for raised liver enzymes increased further. [F. Steffensen et al. Int J Epidemiology (1997) 26(1)].
In another Danish study, self-assessed alcohol intake was determined in a prospective cohort study of 13,285 men and women (aged 30-79 years). The diagnosis of alcohol-induced liver disease was observed. An estimated relative risk of developing liver disease was determined at an intake of 1 - 6 alcoholic beverages per week, with a steep increase in risk above this intake. Women were found to have a significantly higher relative risk of developing alcohol-related liver disease than men. At 7-13 alcoholic beverages per week for women, and 14-27 for men, the relative risk of developing liver disease was greater than one. [U.Becker et al. Hepatology (1996) 23(5)].
An Italian cohort study looked at the prevalence of chronic liver disease. 6534 subjects aged 12-65 were fully examined, and their alcohol intake evaluated with a dietary questionnaire. The risk threshold for developing liver damage was found at ingestion of more than 30gms alcohol/day (both sexes). 21% of the study group were at risk, and 5.5% of this risk group (74 individuals) showed signs of liver damage. Alcoholic cirrhosis was diagnosed in 2.2% of the risk group (ratio men:women, 9:1) and non-cirrhotic liver disease in 3.3%. The authors concluded that in an open population the risk threshold for developing cirrhosis and non-cirrhotic liver damage is 30gms ethanol per day. This risk increases with increased daily intake. [S.Bellentani et al. Gut (1997) 41(6)].
I onda opet dođeš na 80-10-10 koja dokazano ima svojih problema i dugoročno nije dobra ni održiva...
Vrtimo se u krug! Mislim da je važno ne jesti masno i slatko zajedno. Dr Graham je u pravu kad zastupa monomeal - super je voće. Ali nije baš dobro da je SVAKI obrok voće. Tu griješi. I zato mu opada kosa, a koža na vratu ne izgleda ni približno dobro kao kod Lou Corone, na primjer. Kad jedemo masno onda može i bjelančevine, ali ne slatko, a možda čak ni UC općenito! Ogi još ćeš stvoriti novu prehranu, ali moraš dobro povezati sva ta silna istraživanja koja uporno istražuješ.
Pridružen/a: 11. 09. 2007. Postovi: 12892 Lokacija: Zagreb
Postano: 16.9.2015. sri. 13:35 Naslov:
Vrtimo se u krug ali vrti se i cijeli svijet zajedno sa svim doktorima i stručnjacima iz područja prehrane.
Prednost pojedinaca poput mene je u tome što mogu mijenjati mišljenje 5x dnevno.
Ne bi volio biti u koži npr. Aniti Šupe koja kroz knjigu, brojne intervjue i predavanja već godinama uvjerava ljude da je LCHF istina.
Dr. McDougall zagovara sličan omjer kao Graham (nije doktor) ali UH u formi škroba što je logičnije prema mom iskustvu, matematici i povijesti svih velikih cvilizacija.
Mogel bi Ogi napisati knjigu ,,,tak sve pomno bilježiš,,,sigurno bi to pomoglo nekom ,,, A sad onak po mom ,,ova rečenica mi je najbolja,, '' Prednost pojedinaca poput mene je u tome što mogu mijenjati mišljenje 5x dnevno. '' ,,,to je Ogi,,,
Pridružen/a: 11. 09. 2007. Postovi: 12892 Lokacija: Zagreb
Postano: 28.9.2015. pon. 17:26 Naslov:
Citat:
Too little sleep or poor sleep can disrupt your hormones, leading to increased appetite, higher blood glucose, and a thicker waistline. In fact, researchers from the Netherlands found that a single night of sleep deprivation can decrease insulin sensitivity by almost 25 percent.
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